Jack Rivers Auty

Jack completed a Bachelor of Science in anatomy with a neuroscience focus at the southernmost university in the world – the University of Otago. During his degree Jack took several botany papers and fell in love with the subject, so Jack continued studies at Otago with a post graduate diploma in botany followed by a Ph.D. which combined botany and neuroscience by investigating the effects of marijuana-like synthetic cannabinoids on inflammation in the ischemic brain, combining botany and neuroscience.

Jack completed a Bachelor of Science in anatomy with a neuroscience focus at the southernmost university in the world – the University of Otago. During his degree Jack took several botany papers and fell in love with the subject, so Jack continued studies at Otago with a post graduate diploma in botany followed by a Ph.D. which combined botany and neuroscience by investigating the effects of marijuana-like synthetic cannabinoids on inflammation in the ischemic brain, combining botany and neuroscience.

After completing his Ph.D., Jack continued his research at the University of Otago with projects on hydrogen sulphide as an inflammatory signaling molecule and kamikaze neutrophils that release their DNA contents onto bacteria. He then moved to the University of Manchester, where he primarily investigated the role of the inflammasome in Alzheimer’s disease and age-related cognitive decline. Jack was fortunate to have inspiring, supportive, and scientifically proficient supervisors and collaborators in Dr. Catherine Lawrence and Prof. David Brough, under whose guidance he was able to make significant contributions to the field of inflammation research.

Currently, Jack is a lecturer in Medical Sciences at the University of Tasmania, where he investigates inflammation. He is focused on establishing the inflammatory properties of environmental microplastics and identifying the cellular mechanisms that mediate microplastic-induced inflammation. Additionally, he aims to understand how peripheral and central inflammatory responses contribute to Alzheimer’s disease.

For more information checkout www.jackauty.com